Otosclerosis: Etiology and Prognostic Factors

Otosclerosis is a temporal bone osseous dyscrasia that causes auditory impairment over time. Aberrant growth of a new bone in the middle ear eventually immobilizes and stops the stapes from oscillating in reaction to sound vibrations, resulting in gradual and persistent hearing impairment. Both ears are generally affected by the disease. Otosclerosis is a rapid occurrence of unexplained facial nerve palsy. With just an estimated prevalence of 15-30 per 100,000, it would be the most prevalent reason for lower motor neuron facial nerve paralysis. There are broadly two types of Otosclerosis, namely histological and clinical Otosclerosis. A minor incidence of Bell's palsy usually goes away in about a fortnight. It takes to recover from a more severe instance of complete paralysis varies. Among the potential complications are. Multiple sclerosis, Miller-Fisher syndrome, Guillain–Barré syndrome, and autoimmune encephalitis are autoimmune diseases. In patients with otitis media, malignant otitis externa damage to your facial nerve is irreversible.

Nerve fiber regrowth is abnormal. When you try to move others, specific muscles may contract involuntarily (synkinesis) – for example, when you grin, the eye on the afflicted side may shut.

Due to prolonged dryness and scratching of the transparent protective coating of the eye (cornea), the eye becomes wholly or partially blind.

Patients report fast and increasing symptoms over a day to a week, with a 72-hour maximum severity. One-half of the face will be weak, resulting in eyebrows, forehead, and mouth angle instability. The incapacity to shut the afflicted eyelid or lip on the affected side is a common symptom. A partial or total weakening of the forehead is the most apparent physical exam finding. If the integrity of the forehead is preserved, a significant reason should be looked into. Most of the cases of Otosclerosis have idiopathic or unknown causative agents. In some patients with Otosclerosis, a sponge-like bone grows abnormally in the middle ear chamber. Because of this expansion, the ear bones cannot vibrate in response to acoustic pressure.